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This activation process is regulated by several upstream kinases

The former work by exerting high GST-inhibitory activity, while the latter comprise inactive compounds designed to target tumor tissue locally by undergoing GST catalysis in the tumor to release cytotoxic metabolites

Keywords: autoimmune thyroid disease, glp-1 receptor agonists, graves disease, hashimoto thyroiditis, immunometabolism, obesity, semaglutide, thyroid autoimmunity, tirzepatide Introduction and background Introduction Autoimmune thyroid disease (AITD), encompassing Hashimoto thyroiditis and Graves disease, is the most common organ-specific autoimmune disorder, affecting up to 5% of the general population, with a marked female predominance of nearly 5:1 compared with men [1,2]

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